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Ralph E Dolkart</span><span class="contrib-degrees">, M.D.</span>,</li><li><span class="name">Bernard Halpern</span><span class="contrib-degrees">, XX.X.</span>and</li><li><span class="XXXX">XXXXXX XXXXXXX</span><span class="contrib-XXXXXXX">, B.S.</span></li></ol> <h2>Abstract</h2> <p>XXX XXXXXX XXX XXX XXXXXXXXX incidence XXX XXXXXXXX of infections in XXXXXXXX XXXXXXXX has XXXXX been satisfactorily explained. This XXXXXXXXXXXXX was XXXXXXXX XX XXXXX the circulating XXXXXXXX response in XXXXXXX diabetic, XXXXXXX XXXXXXX XXXXXXXX and normal CF-1 XXXX injected with bovine serum albumin. Only those animals XXXXXXX XXXX alloxan who XXX elevated XXXXX glucose levels (XXX XX./XXX ml. or XXXXXX) were XXXXXXXX in XXX study, XXXXXXXX with a group of XXXXXX animals. Animals were bled from XXX XXXXXXX XXXXX XXX XXX XXXXX analyzed for antigen XXXXXXX XXXXXXXX of BSA, glucose XXXXXXXXXXXXX XXX XXXXX proteins. BSA XXX iodinated with I-131 XXX the XXXXXXX XXXXXXX XXXXXXXX of XXXX XXXXX XXXXXX was XXXXXXXXXX as micrograms XX BSA nitrogen bound by 1 ml. XX XXXXXXXXX serum. Our XXXXXXX demonstrate XXXX XXXXX XX XX XXXXXXXXXXX XXXXXXXXXX in XXXXXXXX response of alloxan XXXXXXX mice, alloxan XXXXXXX XXXX given XXXXXXX XXX normal XXXX when XXXXXXXXX XXXX XXX XXXXX the XXXXXXXXXX of XXX XXXXXXXXXXX. If XXX alloxan XXXXXXX XXXXX may XX considered a XXXXXXXXXX model of XXXXXXXX XXXXXXXX in XXX, the results XXXXXXXXX XXXXXX XXXXX XX XXXXXXXXXX XXXX those XXXXXXX XX XXXXXXXXX XXXXX XXXX XXXXX XX primary XXXXXX in XXXXX XXXXXX systems.</p> <XX><li>XXXXXXXXX &XXXX; XXXX by the American Diabetes XXXXXXXXXXX</li></ul>">
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